Vigabatrin associated visual field constriction
نویسندگان
چکیده
منابع مشابه
Visual field defects associated with vigabatrin therapy.
OBJECTIVE To estimate the prevalence of visual field defects in patients taking the anticonvulsant drug vigabatrin and to characterise the features of visual dysfunction found. METHODS Thirty three unselected patients attending neurology and epilepsy clinics were identified as taking vigabatrin and asked to attend for neuro-ophthalmic evaluation. A control group of 16 patients with epilepsy u...
متن کاملVisual field loss associated with vigabatrin: pathological correlations.
Pathological changes are reported in the anterior visual pathways of a 41 year old man with complex partial seizures treated with vigabatrin who developed bilateral visual field constriction. There was peripheral retinal atrophy with loss of ganglion cells and loss of nerve fibres in the optic nerves, chiasm, and tracts. No evidence of intramyelinic oedema was seen. These findings suggest that ...
متن کاملVisual field defect associated with vigabatrin: observational cohort study.
1 Wilkinson RG. Income distribution and life expectancy. BMJ 1992;304:165-8. 2 Wilkinson RG. Unhealthy societies: the afflictions of inequality. London: Routledge, 1996. 3 Kaplan GA, Pamuk E, Lynch JW, Cohen RD, Balfour JL. Inequality in income and mortality in the United States: analysis of mortality and potential pathways. BMJ 1996;312:999-1003. 4 Kennedy BP, Kawachi I, Prothrow-Stith D. Inco...
متن کاملColeman of vigabatrin induced visual field defects
http://bjo.bmj.com/content/86/8/931.full.html Updated information and services can be found at: These include: References http://bjo.bmj.com/content/86/8/931.full.html#ref-list-1 This article cites 3 articles, 2 of which can be accessed free at: service Email alerting the top right corner of the online article. Receive free email alerts when new articles cite this article. Sign up in the box at...
متن کاملGabapentin may cause reversible visual field constriction.
A 52 year old woman presented with neuropathic pain in both legs. Clinical neurological and neurophysiological examinations showed polyneuropathy, and a diagnostic work-up found no evidence of concomitant disorders or current drug use. She was treated with carbamazepin, but we stopped the drug because she had persistent dizziness. We started her on 400 mg gabapentin twice a day, increasing to 8...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: Journal of Neurology, Neurosurgery & Psychiatry
سال: 1999
ISSN: 0022-3050
DOI: 10.1136/jnnp.67.6.707